Role of mitochondrial dysfunction in a model of depression

Research theses
Abstract

The consumption of ultraprocessed food or a high‑fat diet (HFD) has been associated with an increased risk of developing Major Depressive Disorder (MDD), primarily through mechanisms involving neuroinflammation and metabolic dysfunction. Emerging evidence indicates that these effects are sex‑dependent. Here we will study the effect of HFD and chronic stress in the development of a depressive-like phenotype in mice, and the effect on inflammation and mitochondrial dysfunction. In addition, we will analyze the effect of rapid acting antidepressant drugs in this animal model, and the signaling pathways associated to the behavioural effect in male and female animals. This study would allow us to broaden the knowledge associated with difficult to treat depression, and sex‑specific mechanisms.

Keywords
High fat diet
depression
inflammation
mitochondria
sex
ERC sector(s)
PE Physical Sciences and Engineering
Fields of study
Thesis supervisor
Name supervisor
Fuencisla Pilar Cuéllar
E-mail
eunice@unican.es
Department/Faculty/School/Institute/Area/Division NAME
Instituto de Biomedicina y Biotecnología de Cantabria (IBBTEC)
Name of the host University
University of Cantabria (UC)
EUNICE partner e-mail of destination Research
area.eunice@unican.es
Country
Spain
Student profile
Thesis level
Bachelor
Master
PhD
Minimal language knowledge requisite
English B2
Additional info
Length of the research internship
3 months
Financial support available (other than E+)
No
Research interests for cooperation opportunities
Role of neuroplasticity, inflammation and mitochondrial dysfunction in the neurobiology of major depressive disorder and the mechanisms of action of rapid acting antidepressant drugs.